Those of us who have struggled with sugar don’t need a study to tell us that there is something different about sugar than other foods. Dr. Yudkin in the 1970s showed the addictive properties of sugar, and was bold enough to say that had sugar been discovered now it would be a controlled substance. Below is a link to yet another study that lifts up the problem with over use of sugar. While some people can handle sugar and not allow it to become a dependency, just like there are people who can smoke and drink without becoming dependent, there are some people who find they cannot stay away from the powerful draw of sugar. What starts as some overeating can eventually lead to binges, so there is a progressive element to sugar addiction that’s also present in alcohol abuse.
No one likes to think they are addicted to anything, but for those of us who have found ourselves constantly craving more sugar-starchy food even though we have just eaten a big meal know that there is something we have ceased to be able to control when it comes to those highly refined carbohydrates.
Once we accept we can’t have a little and go our merry way, the healing begins. For me it is strict abstaining from sugars, most starches, and artificial sweeteners. Sugar was the only thing in my life I could not control, and while it may not be how I expected to find myself as I aged, at least now there is a plethora of information to support why we struggle with this substance, and why we need to avoid it long before we find ourselves under its control.
Yours in learning and acceptance,
Nan aka Sugarbaby
Here’s the link: http://www.ncbi.nlm.nih.gov/pubmed?Db=pubmed&Cmd=ShowDetailView&TermToSearch=12055324&ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum
Evidence that intermittent, excessive sugar intake causes endogenous opioid dependence.
The goal was to determine whether withdrawal from sugar can cause signs of opioid dependence. Because palatable food stimulates neural systems that are implicated in drug addiction, it was hypothesized that intermittent, excessive sugar intake might create dependency, as indicated by withdrawal signs.
RESEARCH METHODS AND PROCEDURES:
Male rats were food-deprived for 12 hours daily, including 4 hours in the early dark, and then offered highly palatable 25% glucose in addition to chow for the next 12 hours. Withdrawal was induced by naloxone or food deprivation. Withdrawal signs were measured by observation, ultrasonic recordings, elevated plus maze tests, and in vivo microdialysis.
Naloxone (20 mg/kg intraperitoneally) caused somatic signs, such as teeth chattering, forepaw tremor, and head shakes. Food deprivation for 24 hours caused spontaneous withdrawal signs, such as teeth chattering. Naloxone (3 mg/kg subcutaneously) caused reduced time on the exposed arm of an elevated plus maze, where again significant teeth chattering was recorded. The plus maze anxiety effect was replicated with four control groups for comparison. Accumbens microdialysis revealed that naloxone (10 and 20 mg/kg intraperitoneally) decreased extracellular dopamine (DA), while dose-dependently increasing acetylcholine (ACh). The naloxone-induced DA/ACh imbalance was replicated with 10% sucrose and 3 mg/kg naloxone subcutaneously.
Repeated, excessive intake of sugar created a state in which an opioid antagonist caused behavioral and neurochemical signs of opioid withdrawal. The indices of anxiety and DA/ACh imbalance were qualitatively similar to withdrawal from morphine or nicotine, suggesting that the rats had become sugar-dependent.
[PubMed – indexed for MEDLINE]